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Friday, November 3, 2006

On November 13, Torontonians will be heading to the polls to vote for their ward’s councillor and for mayor. Among Toronto’s ridings is Beaches—East York (Ward 32). Four candidates responded to Wikinews’ requests for an interview. This ward’s candidates include Donna Braniff, Alan Burke, Sandra Bussin (incumbent), William Gallos, John Greer, John Lewis, Erica Maier, Luca Mele, and Matt Williams.

For more information on the election, read Toronto municipal election, 2006.

Saturday, April 7, 2007

The former State Lunatic Hospital at Danvers better known as Danvers State Hospital in Danvers, Massachusetts suffered a major fire last night that could be seen for miles and led to temporary shut down of US Route 1 and Interstate 95 in the area.

The infamous American former mental hospital has been closed since 1992 is the site of a new condominium and apartment buliding complex being developed by Avalon Bay Communities. Four apartment units and four utilities bulidings went in up in smoke in the four-alarm fire. The units were not occupied, however, two were nearing completion which is a major financial setback for Avalon Bay. The fire did not spread to the famous Kirkbride building though, which is being renovated into apartments, aside from a cupola due to radiant heat.

Calls started coming just before 2:00 am EDT Saturday morning to report a fire on the hospital’s grounds which before the development sat unused, decaying and abandoned and became a popular target for ghost-hunters, vandals and urban explorers and also served as the focus of the 2001 thriller/horror film, Session 9 which brought renewed attention to infamous hospital which led to increased security at the site over the years. Reports of explosions before the fire started were noted by neighbors and residents.

About 20 residents on the other side of the 77-acre property were evacuated to Danvers High School as a precaution, they are expected to return later today. Their homes were not harmed by the fire, nor were any injuries reported from them. However, about seven firefighters suffered some minor burns from battling the fire, which took seven hours to bring under control. Firefighters will remain on the scene to make sure no hot spots flare up as the fire is still smoldering.

The cause of the fire is not yet known.

Monday, February 11, 2013

With news of Pope Benedict XVI’s resignation only hours old, online gambling sites have already published odds for who will become the next leader of the Roman Catholic Church, with Cardinals Marc Ouellet and Peter Kodwo Appiah Turkson as early odds-on favorites.

Online Australian gambling site SportsBet has Canadian Cardinal Marc Ouellet as their favorite with 7/2 odds. The Irish Paddy Power brokers has 11/4 odds with the Canadian Cardinal being chosen as Pope. Bwin has Ouellet becoming the next Pope with odds at 7/2. British SportingBet also has odds for Ouellet at 7/2.

British Ladbrokes has Nigerian Cardinal Francis Arinze with 7/2 odds.

SkyBet has the betting odd for Ghanian Cardinal Peter Kodwo Appiah Turkson at 7/2, its best odds for any potential Papal candidate. BetVictor also has the best odds on Turkson with 5/2. Stan James has the best odds for Turkson at 3/1. You Win has odds for Turkson at 5/2.

Bets are also being taken by Paddy Power for the name of the next Pope, with Peter their odds-on favorite at 2/1. It is followed by Pious at 5/1, and John Paul and John at 6/1.

Monday, January 18, 2010

The Benet Academy Redwings boys basketball team defeated the Oswego High School Panthers 40–34 Saturday night at Benet’s historic Alumni Gym. 

Located at Lisle, Illinois, United States, the gymnasium is home to Benet’s winning streak of 102 consecutive home games. The statewide record lasted from November 26, 1975 until January 24, 1987, when Naperville North High School defeated the Redwings 47–46. Benet also achieved 96 consecutive victories in the Western Suburban Catholic Conference at that time. 

The school continued to use the facility, colloquially referred to as the “Old Gym” or the “Small Gym”, until the end of the 1994 season, when a newer athletic center was built on campus. It wasn’t until the late 1990’s when then-coach Marty Gaughan decided to play one game per year in the Alumni Gym to remind the school of its history. “There is just an electricity, and you feel it when you are in there,” said Gaughan, who coached the team from the 1989–1990 season to the 2007–2008 season. 

This tradition continued until the 2006–2007 season, when the Redwings played against long-time rival St. Francis High School. Renovations prevented the gym from being used for athletic events over the past two seasons. This game was Benet coach Gene Heidkamp’s first opportunity to coach in the older gym. “So much history and winning has taken place there, which makes it so special. It is something the entire school community is excited about,” said Heidkamp. 

Saturday’s game was played at the Alumni Gym at the request of Oswego assistant coach Jim Bagley. He wanted his son, senior forward Chris Bagley, to experience the same atmosphere he had as he played for Benet in the 1978–1979 season. Greg Kwiatkowski was also Jim Bagley’s teammate in Benet, and his son, Joe Kwiatkowski, was in the Panthers’ starting lineup as well. “Two of us who played at Benet together and now our son’s play on [Oswego] together and for them to have a chance to play where we played is going to be a great night and great experience,” said the senior Bagley. 

Alumni Gym apparently still had its charm in the second half for the Redwings, whose 15–17 score at halftime worsened into an 11-point deficit in the third quarter. Benet fell behind with a score of 21–29 at the start of the fourth quarter. From that point, the Redwings’ defense began to kick in. Oswego could make only 2 of its 13 field goals in the third quarter. Benet’s offense also gained momentum as center Frank Kaminsky scored all of his 9 points in the fourth quarter. A shot by senior Mike Runger brought the score to 31–31, and two consecutive driving layups by David Sobolewski gave Benet the lead that would last for the rest of the game. 

“We hit a dry spell there, but let’s give credit where credit is due. Defense wins games and they are the best defensive team we’ve faced all year,” said Oswego coach Kevin Schnable.

Benet’s Matt Parisi led his team with 15 points, and Sobolewski scored 12. Oswego’s Andrew Ziemnik also scored 12. 

Sunday, June 28, 2020

Last month, a study conducted by archæologist Dr. Naomi Martisius and other researchers concluded Neanderthals living in Europe tens of thousands of years ago were more sophisticated than previously thought. The now-extinct species used to carefully select bones from a particular animal species to manufacture their bone tools, the research showed. The research was published on May 8 in Nature’s Scientific Reports journal.

Dr Martisius and her team used five bone tools discovered from Neanderthals’ sites in southwest France for this research. Four of these bone tools were found in a site called Abri Peyrony and the other one was from Pech-de-l’Azé I. These tools were just a few centimetres in size and were about 50 thousand years old, Dr Martisius told Wikinews. Microscopy analysis of these bone tools called lissoirs (smoothers) suggested Neanderthals used these tools for working animal skin to leathers.

The study stated the fauna of the sites were primarily medium-sized ungulates such as reindeer, in one layer nearly 90%. Despite the overabundance of medium-sized ungulates, Neanderthals used ribs of large bovids for making lissoirs. Dr Martisius told Wikinews this was likely due to the physical characteristics of the bovid ribs, which were “thicker” and “stronger” as compared to the “thin and flimsy ribs” of reindeers. In order to check the origins of the bone tools, the researchers used a technology called non-destructive Zooarchæology by Mass Spectrometry (ZooMS).

Instead of damaging the bone artefacts in order to discover its origins, the researchers collected collagen from the plastic containers in which these artefacts were kept. Collagen is a type of protein. These bone artefacts were kept in plastic containers: some were kept for about five years, some for just a few months. During this time, the collagen proteins from bone tools were stuck to the walls of its plastic containers. The collagen samples collected from the walls of the containers are broken into smaller molecules called peptides by using a chemical enzyme called trypsin.

After the trypsin has broken collagen fibres into peptides, it is analysed using a technology called Matrix-assisted laser desorption/ionization (MALDI) Time-of-Flight mass spectrometer (ToF MS). The assisting matrix is a coloured compound. The acidic peptide is combined with the matrix, vapourised, and peptides are released. Some of them are positively-charged particles which travel across a vacuum tube in an electric field. Depending on the weight of the peptides, these molecules reach the end of the vacuum tube at different instances of time, forming a spectrum. These graphs are like unique fingerprints of a species: they are different for different species of animals. Looking at the database of such graphs, taxonomic identifications of the collagen proteins came be made.

All four bone tools from Abri Peyrony gave positive results and showed that the bones were made from large bovids, even though reindeer were more abundant during that time. One of the advantages of using bovid ribs over reindeer’s thin ribs was the bovid ribs would be more resistant to breaking during flexion, Dr Martisius said.

Dr Martisius said such non-destructive ZooMS analysis was previously conducted, but for tools no older than a few centuries. She said such an analysis had never been previously conducted for artefacts so ancient.

Wikinews caught up with Dr Martisius to discuss this research in-depth.

18 December 2004

Emergency hospital during 1918 influenza epidemic, Camp Funston, Kansas (source: National Museum of Health and Medicine, AFIP).

A bout of the flu can be mild. In young, healthy adults, many infections pass unnoticed. But sometimes the influenza virus evolves into a strain that decimates its victims. The worst known strain swept the world in the Fall of 1918, infecting 500-1000 million and killing 40-100 million, about 2-5% of people.

There are several theories about where the pandemic began, but the likeliest origin was in Haskell County, Kansas, in the United States. People in the sparsely populated county, where farmers raised pigs, poultry, cattle, and grain, began suffering from influenza in late January 1918. Unusually for flu, it was young, healthy adults who were hardest hit. Victims fell ill suddenly, many progressing to pneumonia and dying, often within days. Within weeks, however, the epidemic ended. The natural geographic isolation of this community normally might have contained the fatal flu in a sort of unintentional quarantine, but the First World War intervened. Men were uprooted from their home towns and congregated in huge numbers in army camps for training and then shipping out to other camps or to fight in Europe. The destination for men from Haskell County was Camp Funston, part of Fort Riley, Kansas, where the first influenza case was reported in early March. As soldiers moved among camps, the virus spread. Within two months, the epidemic spread to most of the army camps and most of the largest cities in the United States. As American soldiers went to France, so did the virus, spreading first from the port of Brest.

The flu then spread worldwide. The pandemic reached its height in the Fall of 1918. Spain was affected early, and because Spain was not fighting in the World War, there was no wartime censorship, and news of the outbreak became widely known, leading to the flu being called the Spanish Flu in many countries. In Spain, however, it was called French Flu or the Naples Soldier. In India, about 12 million people died of flu. In some US cities, people died so quickly that morticians couldn’t cope with the bodies. According to Jessie Lee Brown Foveaux, who worked in the Fort Riley laundry during the epidemic: “They were piling them up in a warehouse until they could get coffins for them.”

The disease started with cough, then headache. Temperature, breathing and heart rate increased rapidly. In the worst cases, pneumonia came next, the lungs filling with liquid, drowning the patients and turning them blue from lack of air. Patients bled from every orifice: mouths, noses, ears, eyes. Those who survived often suffered temporary or permanent brain damage. Several million developed encephalitis lethargica, in which victims were trapped in a permanent sleeplike and rigid state, as portrayed in the 1990 movie “Awakenings.” In others, normal thought processes were impaired. During negotiations to end World War I, US President Woodrow Wilson was struck with flu, and people around him noted that his mental abilities never fully recovered. The French leader George Clemenceau had wanted harsher punishment of Germany than Wilson had desired. Clemenceau may have convinced Wilson in his weakened state to accept such harsh terms, which may have been one of the factors causing World War II.

Since flu is highly contagious early in the illness, even before symptoms appear, strict quarantine may be necessary to stop its spread during an epidemic. Australia kept its 1918 flu death rate relatively low by enforcing quarantines. However, in many parts of the world, public health officials hesitated to impose such measures, giving the disease time to gain a foothold. In the US city of Philadelphia, a rally of half a million people was planned in September 1918 to sell bonds to fund the war, at just the time when the flu started to infect residents. Although doctors warned the public health director to cancel the rally, he wanted to meet the city’s quota to raise money for the war and refused to cancel the event. Within days after the rally, half a million city residents caught the flu.

Why was the 1918 flu so deadly? The influenza virus wasn’t preserved at the time of the outbreak, at least on purpose. But in the late 1990s researchers Ann Reid, Jeffery K. Taubenberger, and their colleagues extracted and sequenced the genetic material of the virus, RNA, from tissue of victims who died in the pandemic. They used bits of lung that were preserved in formalin from victims on army bases or from victims buried in permafrost in the Alaskan village of Brevig Mission, where flu killed 85% of adults. Comparisons with known flu viruses in humans, pigs, and birds suggest that some genes of the 1918 virus came from birds or an unknown animal source. Other scientists then were able to show that the amino acid sequence of hemagglutinin protein from the 1918 virus had several changes from other flu viruses that may have helped it to easily bind and invade human cells, and that made the virus look different enough from earlier flu virus strains that people had no immunity.

The possibility exists that another flu pandemic will sweep the world like the one in 1918. In 2004, an H5N1 influenza virus has killed millions of birds and at least 30 people in southeast Asia. So far this virus strain has not evolved the ability to pass directly from human to human, but that possibility becomes more likely as the bird flu pandemic continues and humans remain in contact with chickens, ducks, and other birds. The virus has killed two-thirds of people reported to be infected. Dr. Tim Uyeki, an epidemiologist for the US Centers for Disease Control, says, “you have the ingredients in Asia right now for a public health disaster.”

But since sequences of this bird flu virus are known, it may be possible to develop a vaccine or set of vaccines to protect against it. At a special meeting of influenza experts on November 11th and 12th, World Health Organization influenza program chief Klaus Stohr said, “It is not only possible, but also important, that influenza pandemic vaccines be made available… and there’s a shared responsibility needed to make that happen…. We have a huge window of opportunity now.”